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Conduct a survey. Advise you on how to care for the sick. Prescribe appropriate treatment or refer to a neurologist, psychiatrist, gerontologist and other specialists for further examination and treatment. Recently, new drugs have been developed that can help halt the progression of Alzheimer's disease. Alzheimer's disease is a progressive form of senile dementia, leading to a complete loss of cognitive abilities, developing mainly after 60-65 years of age. It is clinically manifested by a gradual and constantly progressive disorder of cognitive abilities: attention, memory, speech, praxis, gnosis, psychomotor coordination, orientation and thinking. Diagnosis of atorvastatin online disease allows a thorough history taking, PET of the brain, exclusion of other types of dementia using EEG, CT or MRI. Treatment is palliative, including medical (cholinesterase inhibitors, memantine) and psychosocial (art therapy, psychotherapy, sensory integration, simulation of presence) therapy.

Causes Symptoms of Alzheimer's disease Predementia Early dementia Moderate dementia Severe dementia Diagnosis Treatment of Alzheimer's disease Prognosis and prevention Prices for treatment. Alzheimer's disease got its name from the name of the German psychiatrist who first described it in 1906. The incidence on average ranges from 5 to 8 people. per 1000 population, which is about half of all cases of diagnosis of dementia. On a global scale, according to 2006, the number of patients with Alzheimer's disease was 26.5 million people. There is a clear trend towards an increase in the incidence, which makes the problem of diagnosing and treating this pathology one of the important tasks of modern psychiatry and neurology.

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Make sure that the elderly in your family have regular medical check-ups. Seek immediate medical attention if you suspect that you or anyone in your family has symptoms of Alzheimer's disease.

There is a significant correlation between the incidence of Alzheimer's dementia and age. Thus, in the age group of 65 years, there are about 3 cases of the disease per 1000 people, and among people aged 95 years there are already 69 cases per 1000. The prevalence of lipitor pills in developed countries is much higher, since their population has a longer life expectancy. Alzheimer's disease is more common among women than among men, which is partly attributed to their higher life expectancy compared to men.

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An older theory is the cholinergic theory of the onset of the disease, linking it to acetylcholine deficiency. However, the results of clinical studies have shown the inability of acetylcholine preparations to at least partially or temporarily stop Alzheimer's disease. The amyloid hypothesis of the development of the disease has existed since 1991.

According to it, accumulations of beta-amyloid are the basis of the pathology. Interestingly, the gene encoding the beta-amyloid precursor protein is part of chromosome 21, the trisomy of which underlies Down syndrome. At the same time, all patients with Down syndrome who have reached the age of 40 have an Alzheimer-like pathology.

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The predisposing factors for the synthesis of pathological beta-amyloid are the insufficiency of mitochondrial oxidation processes, a more acidic reaction of the intercellular environment, and an increased amount of free radicals. Pathological amyloid deposits are noted both in the brain parenchyma and in the walls of Atorvastatin vessels. It should be noted that such deposits characterize not only Alzheimer's disease, they are observed in cerebral hematomas of congenital origin, Down's syndrome, and during normal aging processes.

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According to the third hypothesis, Alzheimer's disease is associated with the death of neurons as a result of the accumulation of hyperphosphorylated tau protein in them, the threads of which stick together and form tangles. According to the tau hypothesis, protein accumulation is associated with a defect in its structure; the formation of plexuses causes the disintegration of intraneuronal transport, which in turn leads to disruption of signal transmission between neurons, and then to their destruction.

 

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Therefore, many researchers deny the independent pathogenetic significance of the tau protein, considering its accumulation as a consequence of the mass destruction of neurons.

On the other hand, the formation of atorvastatin tangles is also observed in other cerebral degenerations (for example, in progressive supranuclear palsy and frontotemporal atrophy). Possible Causes of Alzheimer's Disease, is called the synthesis of pathological apolipoprotein E. The latter has an affinity for the amyloid protein and is involved in the transport of the tau protein, which may underlie the typical morphological changes of the disease described above.

In typical cases, Alzheimer's disease manifests itself in people older than 60-65 years.

According to many researchers, Alzheimer's disease is genetically determined. Five main genetic regions with which the development of the disease is associated have been identified. They are located on chromosomes 1, 12, 14, 19 and 21. Mutations in these loci lead to disturbances in the protein metabolism of cerebral tissues, resulting in the accumulation of lipitor or tau protein. Symptoms of Alzheimer's disease.

It is extremely rare to find cases of an early form of the disease that occur between 40 and 60 years of age. Dementia of the Alzheimer's type is characterized by a subtle and prolonged onset, steady progression without periods of improvement.

The main substrate of the disease are disorders of higher nervous functions. The latter include: short-term and long-term memory, mindfulness, spatial and temporal orientation, psychomotor coordination (praxis), the ability to perceive various aspects of the external world (gnosis), speech, control and planning of higher neuropsychic activity. Alzheimer's disease is divided into 4 clinical stages: predementia, early, moderate and severe dementia.

The speech of patients is reduced to the use of separate phrases or single words. In the future, speech skills are completely lost. At the same time, the ability to perceive and maintain emotional contact with others is preserved for a long time. Alzheimer's disease in the stage of severe dementia is characterized by complete apathy, although aggressive manifestations can sometimes be observed.

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One of the main areas of diagnostic search is the collection of anamnesis and complaints. Since the patient himself in the early stages of the disease often does not notice the changes happening to him, and with the development of dementia he cannot adequately assess his condition, the survey should be conducted among his relatives.

Diagnose pre-stage Alzheimer's diseaseemences is quite difficult. During this period, only advanced neuropsychological testing can reveal some impairment of higher nervous functions. During the study, patients are asked to memorize words, copy figures, perform complex arithmetic operations, read and retell what they read.

In order to exclude other diseases that can lead to the development of dementia, a neurologist conducts a neurological examination, prescribes additional examinations: EEG, REG, Echo-EG, CT or MRI of the brain. Of certain importance in confirming the diagnosis is the detection of beta-amyloid deposits during PET of the brain with the introduction of Pittsburgh composition B. Recently, it has been proven that another marker of the disease can be the detection of tau protein or beta-amyloid in cerebrospinal fluid taken for analysis by lumbar punctures.

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Differential diagnosis of dementia of the Alzheimer's type is carried out with vascular dementia, parkinsonism, dementia with Lewy bodies, dementia in epilepsy, and other neurological pathologies. Treatment of Alzheimer's disease.

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